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1.
Postnatal Ozone Exposure Disrupts Alveolar Development, Exaggerates Mucoinflammatory Responses, and Suppresses Bacterial Clearance in Developing Scnn1b-Tg+ Mice Lungs.
J Immunol
; 207(4): 1165-1179, 2021 08 15.
Article
in English
| MEDLINE | ID: mdl-34330754
2.
The Innate Lymphoid System Is a Critical Player in the Manifestation of Mucoinflammatory Airway Disease in Mice.
J Immunol
; 205(6): 1695-1708, 2020 09 15.
Article
in English
| MEDLINE | ID: mdl-32817334
3.
Compartment-specific transcriptomics of ozone-exposed murine lungs reveals sex- and cell type-associated perturbations relevant to mucoinflammatory lung diseases.
Am J Physiol Lung Cell Mol Physiol
; 320(1): L99-L125, 2021 01 01.
Article
in English
| MEDLINE | ID: mdl-33026818
4.
Ozone exposure upregulates the expression of host susceptibility protein TMPRSS2 to SARS-CoV-2.
Sci Rep
; 12(1): 1357, 2022 01 25.
Article
in English
| MEDLINE | ID: mdl-35079032
5.
Vesicular and extravesicular protein analyses from the airspaces of ozone-exposed mice revealed signatures associated with mucoinflammatory lung disease.
Sci Rep
; 11(1): 23203, 2021 12 01.
Article
in English
| MEDLINE | ID: mdl-34853335
6.
Myeloid-IL4Rα is an indispensable link in IL-33-ILCs-IL-13-IL4Rα axis of eosinophil recruitment in murine lungs.
Sci Rep
; 11(1): 15465, 2021 07 29.
Article
in English
| MEDLINE | ID: mdl-34326406
7.
Ozone exposure upregulates the expression of host susceptibility protein TMPRSS2 to SARS-CoV-2.
bioRxiv
; 2020 Nov 11.
Article
in English
| MEDLINE | ID: mdl-33200134
8.
Paradoxical reaction to antitubercular treatment in a case of pulmonary tuberculosis.
BMJ Case Rep
; 20162016 Feb 17.
Article
in English
| MEDLINE | ID: mdl-26887885
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